波士顿麻省总医院肿瘤抑制分子机制研究博士后岗位
日期：2008-04-22
来源：丁香人才网
InstitutionMassachusetts General Hospital (http://www.partners.org)PositionPostdoctoral FellowLocationBoston MADate PostedApril 21th 2008Date ExpiresJune 30th 2008Tagspostdoctoral fellow ma usa and Bo ...
Institution
Massachusetts General Hospital (http://www.partners.org)
Position
Postdoctoral Fellow
Location
Boston, MA
Date Posted
April 21th 2008
Date Expires
June 30th 2008
Tags
postdoctoral fellow, ma, usa, and Boston
Description
A postdoctoral fellowship is available in the Neuroendocrine Unit at theMassachusetts General Hospital and Harvard Medical School to study themolecular mechanisms by which MEG3, a non-coding RNA suppresses humantumor growth and regulates differentiation and development ofneuroendocrine cells and tissues (Zhou et al. Activation of p53 by MEG3non-coding RNA. J Biol Chem. 2007; 282: 24731–42). We are employingbiochemistry, molecular biology, cell biology, and mouse knockout modelsto analyze the functions of MEG3. Applicants must have received theirPh.D. degree within the last 3 years and demonstrate a strong backgroundin cell/molecular biology.
Contact
Experience with mouse knockout/transgenic models a plus. UScitizen/permanent resident preferred. Send CV and references to: Dr.Anne Klibanski (aklibanski@partners.org) or Dr. Xun Zhang(xzhang5@partners.org.),
Neuroendocrine Unit, Massachusetts General Hospital and Harvard MedicalSchool, 55 Fruit Street, BUL457, Boston, MA 02114. MGH is an EqualOpportunity Employer.
Massachusetts General Hospital (http://www.partners.org)
Position
Postdoctoral Fellow
Location
Boston, MA
Date Posted
April 21th 2008
Date Expires
June 30th 2008
Tags
postdoctoral fellow, ma, usa, and Boston
Description
A postdoctoral fellowship is available in the Neuroendocrine Unit at theMassachusetts General Hospital and Harvard Medical School to study themolecular mechanisms by which MEG3, a non-coding RNA suppresses humantumor growth and regulates differentiation and development ofneuroendocrine cells and tissues (Zhou et al. Activation of p53 by MEG3non-coding RNA. J Biol Chem. 2007; 282: 24731–42). We are employingbiochemistry, molecular biology, cell biology, and mouse knockout modelsto analyze the functions of MEG3. Applicants must have received theirPh.D. degree within the last 3 years and demonstrate a strong backgroundin cell/molecular biology.
Contact
Experience with mouse knockout/transgenic models a plus. UScitizen/permanent resident preferred. Send CV and references to: Dr.Anne Klibanski (aklibanski@partners.org) or Dr. Xun Zhang(xzhang5@partners.org.),
Neuroendocrine Unit, Massachusetts General Hospital and Harvard MedicalSchool, 55 Fruit Street, BUL457, Boston, MA 02114. MGH is an EqualOpportunity Employer.
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